Drugs online research references
Jugosl Ginekol Opstet. 1983 May-Aug;23(3-4):61-4.
[Effect of histamine and its antagonists on the muscular tone of the non-pregnant uterus in the woman]
[Article in Croatian]
Atanackovic D, Frkovic A, Frkovic A, Mirosevic L.
In the isolated strips of the individual parts of the human uterus (body, corns, cervix) histamine, promethazine, and cimethidine, proportionally to the dose applied, have proved to increase the muscular tonus. The blocking agents of histamine H-1 receptors (promethazine) and H-2 receptors (cimetidine), separately or jointly administered in the dose of 10 micrograms/ml, significantly diminish the tonicizing effect of histamine. The effect of histamine on the uterine muscular tonus is brought about through histamine H-1 and histamine H-2 receptors.
online pharmacy ref source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=6664101&dopt=Abstract
Jugosl Ginekol Opstet. 1983 May-Aug;23(3-4):65-8.
[Effect of histamine and its antagonists on the muscle tone of the body of the non-pregnant and pregnant human uterus]
[Article in Croatian]
Atanackovic D, Frkovic A, Frkovic A, Mirosevic L.
In the uterus body of nongravid and gravid women, proportionally to the dose used, histamine, promethazine, and cimetidine produce an increased muscular tonus, which is more pronounced in gravid uteri. The blocking agent of histamine H-1 receptors, promethazine, and the blocking agent of histamine H-2 receptors, cimetidine, separately or jointly applied in a dose of 10 micrograms/ml, significantly decrease the tonicizing effect of histamine. Histamine exerts its influence on the muscular tonus of the body of nongravid and gravid human uteri through histamine H-1 and histamine H-2 receptors.
online pharmacy ref source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=6664102&dopt=Abstract
Arch Biochem Biophys. 1984 Feb 1;228(2):450-9.
Mechanisms of the killing of cultured hepatocytes by hydrogen peroxide.
Rubin R, Farber JL.
Mechanisms of H2O2-induced cell injury were explored in primary cultures of rat hepatocytes. Cells prepared from male rats and cultured for 1 day prior to treatment were killed by H2O2 either added directly to the medium at 0.25-2 mM or generated in situ by glucose oxidase (0.25-2 U/ml) or xanthine oxidase (20-120 mM/ml) and 2 mM xanthine. Catalase protected the cells in each case. Lipid peroxidation as measured by the accumulation of malondialdehyde (MDA) preceded the cell death due to H2O2 added directly to the cultures or generated in the medium. The antioxidants N,N'-diphenyl-p-phenylenediamine (DPPD) and promethazine prevented the accumulation of MDA in both cases and protected the cells treated with H2O2 directly. DPPD and promethazine did not react directly with H2O2. Other antioxidants including butylated hydroxytoluene, vitamin E, and N-propylgallate had varied protective activity against the addition of H2O2 in proportion to their ability to reduce MDA accumulation. In glucose oxidase-treated cultures, DPPD and promethazine prevented the cell killing during the first hour but failed to protect between 1 and 3 h despite prevention of lipid peroxidation. The cell killing between 1 and 3 h in the presence of DPPD was prevented by catalase indicating its dependence upon continued generation of H2O2. Further addition of H2O2 in the presence of DPPD also increased the number of dead cells without lipid peroxidation. The data are consistent with at least two mechanisms of hepatocyte killing by H2O2. The first pathway is prevented by the antioxidants DPPD and promethazine and is very likely related to the peroxidation of membrane phospholipids. The second is independent of lipid peroxidation yet dependent upon the continued presence of H2O2.
online pharmacy ref source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=6696441&dopt=Abstract
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