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J Affect Disord. 1979 Dec;1(4):227-35.
Evidence that piracetam has an anxiolytic action.

File SE, Hyde JR.

In the social interaction test of anxiety Piracetam (100 mg/kg) had an anxiolytic profile very similar to that seen after 5 days of administration of chlordiazepoxide (5 mg/kg). Piracetam (50-300 mg/kg) produced no signs of sedation and it was therefore suggested that it might be a non-sedative anxiolytic drug. Piracetam (100 mg/kg) produced significantly higher cortical concentrations of 5-hydroxytryptamine and lower concentrations of 5-hydroxyindoleacetic acid, indicating a reduced 5-HT turnover. There were no drug-induced changes in noradrenaline or dopamine in any brain region, either with or without pretreatment with alpha-methylparatyrosine. The cortical concentrations of 7 amino acids were measured and were unchanged by treatment with Piracetam.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=95599&dopt=Abstract

Percept Mot Skills. 1978 Apr;46(2):467-75.
Suppression of visual observing by rhesus monkeys produced by conditioned aversive visual stimuli.

Kremer ME, Napierala JS, Haude RH.

Nine rhesus monkeys were tested in a visual observing situation to determine the influence of conditioned aversive visual stimulation. A set of meaningless visual stimuli was selected on the basis of cumulative frequency and cumulative duration of observing during a pretest phase of the experiment. Three subsets of stimuli (low, medium, high) were formed indicating level of observing during pretesting. A portion of the "medium" category of slides then served as conditioned stimuli in a classical conditioning procedure by being paired with electric shock. Following conditioning the entire set of stimuli was again presented in a visual observing situation. The results showed a significant decrease in both frequency and duration of observing of the slides with conditioned aversive qualities, relative to non-shock control slides. These findings support an aversion-produced suppression of observing relatively unconfounded by methodological, procedural, and other differences existing among previous reports on the role of fear or anxiety in this context.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=96417&dopt=Abstract

Arch Int Pharmacodyn Ther. 1978 Jun;233(2):326-42.
Pharmacological profile of N-(2,6-DIMETHYLPHENYL)-N-(1-methyl-2pyrrolidinylidene)urea, xilobam, a new centrally acting skeletal muscle relaxant.

Gardocki JF, Hageman WE, Pruss TP.

The pharmacological profile of a new centrally acting skeletal muscle relaxant, xilobam, is described and compared to that of existing skeletal muscle relaxants. The potencyof xilobam, administered intravenously, is approximately ten times that of methocarbamol in the linguomandibular assay in the cat. When evaluated in the strychnin assay in the mouse, the potency of xilobam is approximately seven times that of chlorzoxazone and eleven tomes that of methocarbamol and five times that of metaxalone. In contrast to methocarbamol, xilobam exhibits little or no sedative activity and appears devoid of antianxiety properties. When administered to non-anesthetized dogs, xilobam and other centrally acting muscle relaxants, such as chlorzoxazone and methocarbamol, increased arterial pressure and heart rate. Mydraiasis, vocalization and muscle rigidity were concomitantly observed. These effects appear to be centrally induced. It is concluded that xilobam appears to be a potent centrally acting muscle relaxant which should not be sedating or anxiolytic in man.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=99099&dopt=Abstract

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