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Alopecia, hair loss abstracts ||
J Dermatol. 2002 Oct;29(10):653-6.
Idiopathic CD4+ T lymphocytopenia associated with disseminated flat warts and alopecia areata.
Gubinelli E, Posteraro P, Girolomoni G.
Istituto Dermopatico dell'Immacolata, IRCCS, Rome, Italy.
Idiopathic CD4+ T lymphocytopenia is a very rare condition characterized by persistent depletion of circulating CD4+ T lymphocytes, without evidence of HIV or HTLV infection, or other identifiable causes of immunodeficiency. The syndrome can present with dermatological diseases, including viral, fungal and bacterial infections, as well as Kaposi's sarcoma, epithelial cell malignancies, lymphoma and inflammatory dermatoses. We report the case of a 47-year-old woman with idiopathic CD4+ T lymphocytopenia who presented with a 10-year history of disseminated and refractory flat warts from which human papillomavirus type 3 DNA was identified. The patient also had alopecia areata.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12432998&dopt=Abstract
Nouv Presse Med. 1978 Jan 7;7(1):29-34.
[Sezary's syndrome (author's transl)]
[Article in French]
Guilhou JJ, Meynadier J, Clot J.
The syndrome described in 1938 by Sezary and Bouvrain is characterized by a possibly hyperpigmented erythroderma with oedema and infiltration of the skin, palmo-plantar keratoderma, diffuse alopecia and large lymphadenopathies. The cutaneous histopathology frequently shows a dermal mononuclear infiltrate with, sometimes, epidermal microabcesses. But none of these signs is actually specific for the Sezary syndrom, the only criteria of which is the presence of circulating Sezary cells with their folded, cerebriform nucleus demonstrated by electron microscopy. The Sezary cell is to date clearly identified as a T lymphocyte but membrane markers and Tcell fonction studies could elicite abnormal and poor reactive T cell. In order to assert the Sezary Syndrome it is stated by the authors that the erythroderma must be associated with more than 10% of Sezary cells in peripheral blood. This feature is needed to differentiate the Sezary syndrome from the erythrodermic form of mycosis fondoides in which the abnormal T cell proliferation is mainly located in the skin. The relationship with the T cell chronic lymphatic leukemia and the main treatments of the Sezary syndrome are discussed.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=149300&dopt=Abstract
Rheumatology (Oxford). 2000 Jun;39 Suppl 1:48-56.
Efficacy and safety of leflunomide in active rheumatoid arthritis.
Smolen JS, Emery P.
Division of Rheumatology, University of Vienna, Austria.
Leflunomide, the newest disease-modifying anti-rheumatic drug (DMARD) for rheumatoid arthritis (RA), inhibits de novo pyrimidine biosynthesis. In two placebo-controlled trials, leflunomide was superior to placebo and comparable to sulphasalazine and methotrexate for improving the signs and symptoms of RA, significantly improved functional ability compared with placebo, sulphasalazine and methotrexate, and was superior to methotrexate and comparable to sulphasalazine in slowing radiographically assessed disease progression. In a multinational trial vs methotrexate, leflunomide showed an American College of Rheumatology (ACR) response rate similar to that in placebo-controlled trials. The ACR response rate with methotrexate was significantly greater. Differences between these trials included methotrexate dosing regimens, folate usage and disease duration. Common adverse events with leflunomide included gastrointestinal symptoms, allergic reactions, alopecia and elevated liver enzyme levels. No long-term safety issues were reported with leflunomide at 2 yr. Efficacy of leflunomide was maintained at 2 yr. Leflunomide is a safe and efficacious addition to the roster of anti-rheumatic drugs.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=11001380&dopt=Abstract
Since hair growth is a complicated biological process, modern science has yet to grasp a complete picture. A number of traditional and alternative therapeutic methods that include drugs, surgery, and suppelements have been developed to help those who are losing hair. Unfortunately, none of these approaches are perfect for all hair loss problems due to the complexity of the phenomenon and diverse nature of the causes underlying hair loss. Also, most of chemical drugs and hair transplantation surgeries are accompanied by undesirable side effects.
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